growth factor-mediated cell survival, cancer biology, transcription network mapping, gene regulation, neuroscience, endocrinology, neuroendocrinology, physiology, carcinogenesis, cell biology, molecular biology, transcription regulation

J.W. Tullai†, M.E. Moss, S.M. Sepulveda, J.F. Brennan, F.J. Naya and G.M. Cooper, (2016) Role of GSK-3 in CREB-mediated transcription regulation, hypertrophy and survival in cardiomyocytes. In revision. PLoS ONE. †Corresponding Author J.W. Tullai, J.R. Graham, G.M. Cooper (2011) A GSK-3-mediated transcription network maintains repression of immediate early genes in quiescent cells. Cell Cycle. 10 (18): 3072-3077. Review Article. J.W. Tullai, L. J. Owens, S. Tacheva, J.R. Graham, and G.M. Cooper (2011) AP-1 is a Component of the Transcriptional Network Regulated by GSK-3 in Quiescent Cells. PLoS ONE. 6(5): e20150. J.R. Graham, J.W. Tullai and G.M. Cooper (2010) GSK-3 represses growth factor-inducible genes by inhibiting NF-kB in quiescent cells. J. Biol. Chem. 285: 4472-4480. J. Terragni, J. R. Graham, M.E. Schaffer, J.W. Tullai, and G.M. Cooper. (2008) The roles of forkhead and NF-kB in the transcription regulation of the phosphatidylinositol 3-kinase pathway. BMC Cell Biol. 9: 6. J.W. Tullai, M.E. Schaffer, S. Mullenbrock, G. Sholder, S. Kasif and G.M. Cooper. (2007) Immediate-early and delayed primary response genes are distinct in function and genomic architecture. J. Biol. Chem. 282: 23981-95. J.W. Tullai, J. Chen, M.E. Schaffer, E. Kamenetsky, S. Kasif, and G.M. Cooper. (2007) Glycogen synthase kinase-3 represses cyclic AMP response element-binding protein (CREB)-targeted immediate early genes in quiescent cells. J. Biol. Chem. 282: 9482-91. J.W. Tullai, M.E. Schaffer, S. Mullenbrock, S. Kasif and G.M. Cooper. (2004) Identification of transcription factor binding sites upstream of human genes regulated by the phosphatidylinositol 3-kinase and MEK/ERK signaling pathways. J. Biol. Chem. 279: 20167-77.