教育经历 1982 年 - 1986 年，就读于武汉大学，获学士学位。 1986 年 - 1989 年，在武汉大学继续深造，获硕士学位。 1999 年 - 2000 年，于武汉大学攻读并获得博士学位。 工作经历 1989 年 7 月 - 1999 年 8 月，在湖北省生物工程研究所工作。 2000 年 7 月 - 至今，在武汉大学生命科学学院任职。

昆虫病毒与宿主相互作用

代表性学术论文（* ：通讯作者） Qiao J and Liu Q*. (2022) Interplay between autophagy and Sindbis virus in cells derived from key arbovirus vectors, Aedes albopictus and Aedes aegypti mosquitoes. Cellular Signalling 90:110204. Wang Y, Qiao J, Zhang D, Zhong C, Wang S, Li X, Feng L, Shi S, Wang B, Liu Q*. (2021) Systematic identification of autophagy-related proteins in Aedes albopictus. PLoS ONE 16(1):e0245694. Qiao J, Zhang D, Wang Y, Li X, Wang S, Qingzhen Liu*. (2018) Identification of AaAtg8 as a marker of autophagy and a functional autophagy-related protein in Aedes albopictus. PeerJ 6:e5988. Yang Z, Zhou K, Liu H, Wu A, Mei L, Liu Q*. (2016) SfDredd, a Novel initiator caspase possessing activity on effector caspase substrates in Spodoptera frugiperda. PloS ONE 11(3):e0151016. Meng, K., Li, X., Wang, S., Zhong, C., Yang, Z., Feng, L., and Liu, Q*. (2016) The Strica homolog AaCASPS16 is involved in apoptosis in the yellow fever vector, Aedes albopictus. PloS ONE 11(6):e0157846. Li X, Meng K, Qiao J, Liu H, Zhong C, Liu Q*. (2016) Identification of Aadnr1, a novel gene related to innate immunity and apoptosis in Aedes albopictus. Gene 587(1):18-26. Feng, L., Liu, H., Li, X., Qiao, J., Wang, S., Guo, D., and Liu, Q*. (2016) Identification of AaCASPS7, an effector caspase inAedes albopictus. Gene 593(1):117-25. Deng, Z., Yan, H., Hu, J., Zhang, S., Peng, P., Liu, Q* and Guo, D*. (2012) Hepatitis C virus sensitizes host cells to TRAIL-induced apoptosis by up-regulating DR4 and DR5 via a MEK1-dependent pathway. PloS ONE 7(5):e37700. Sin O, Mabiala P, Liu Y, Sun Y, Hu T, Liu Q* and Guo D. (2012) Gene silencing efficiency and INF-beta induction effects of splicing miRNA 155-based artificial miRNA with pre-miRNA stem-loop structures. Biochemical genetics 50(1-2):112-21. Yang W, Qu S, Liu Q*, Zheng C*. (2009) Avian influenza virus A/chicken/Hubei/489/ 2004 (H5N1) induces caspase-dependent apoptosis in a cell-specific manner. Mol Cell Biochem 332(1-2):233-41. Liu Q* and Chejanovsky N*. (2006) Activation pathways and signal-mediated upregulation of the insect Spodoptera frugiperda caspase-1. Apoptosis 11(4):487-96. Liu Q, Clem RJ*. (2011) Defining the core apoptosis pathway in the mosquito disease vector Aedes aegypti: the roles of iap1, ark, dronc, and effector caspases. Apoptosis 16(2):105-13. Liu Q, Qi Y and Chejanovsky N* (2005) Spodoptera littoraliscaspase-1, a Lepidopteron effector caspase inducible by apoptotic signaling. Apoptosis 10(4):787-795. Liu Q, Qi Y, Chejanovsky N* (2003) Identification and classification of the Spodoptera littoralis nucleopolyhedrovirus inhibitor of apoptosis gene. Virus Genes 26 (2):143-149. Bryant B, Ungerer MC, Liu Q, Waterhouse RM, Clem RJ*. (2010) A caspase-like decoy molecule enhances the activity of a paralogous caspase in the yellow fever mosquito,Aedes aegypti. Insect Biochem Mol Biol. 40(7):516-23. Li J, Ishaq M, Prudence M, Xi X, Hu T, Liu Q, Guo D*. (2009) Single mutation at the amino acid position 627 of PB2 that leads to increased virulence of an H5N1 avian influenza virus during adaptation in mice can be compensated by multiple mutations at other sites of PB2. Virus Res. 144(1-2):123-9. Wu X, Zhou Y, Zhang K, Liu Q, Guo D*. (2008) Isoform-specific interaction of pyruvate kinase with hepatitis C virus NS5B. FEBS Lett. 582 (15):2155-60. Ishaq M, Hu J, Wu X, Fu Q, Yang Y, Liu Q, Guo D*. (2008) Knockdown of cellular RNA helicase DDX3 by short hairpin RNAs suppresses HIV-1 viral replication without inducing apoptosis. Mol Biotechnol. 39(3):231-8. Li J, Cai H, Liu Q, Guo D*. (2008) Molecular and pathological characterization of two H5N1 avian influenza viruses isolated from wild ducks. Virus Genes. 37(1):88-95. Chen P, Jiang M, Hu T, Liu Q, Chen XS, Guo D*. (2007) Biochemical characterization of exoribonuclease encoded by SARS coronavirus. J Biochem Mol Biol. 40(5):649-55. Kuang E, Wan Q, Li X, Xu H, Liu Q, Qi Y*. (2005) ER Ca2+ depletion triggers apoptotic signals for endoplasmic reticulum (ER) overload response induced by overexpressed reticulon 3 (RTN3/HAP). J Cell Physiol. 204(2):549-59.